Whether you live in an apartment or a house or just need to ensure that your house is free from EMFs, there are a number of ways you can reduce exposure. One of the most straightforward is to limit the use of electronic devices. You could also consider EMF block paint to stop EMF radiation from entering your home. Another way to shield your home from EMF radiation is to use a RF shielding canopy. It is a type of net that has EMF shielding. It is utilized to stop EMFs from entering a room. Another alternative is to have your home fitted with a conductive enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This may account for the long-term effects observed in certain patients suffering from advanced HCC. However, the reason behind AM EMF's impact on patients with cancer is not yet clear.
Aspects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. First, emf blocking that was unaffected RF EMF. The second group was exposed to RF EMF at the same frequency to the frequency used by humans. In block emf , they were subjected the RF EMF in HCC-specific frequencies. The effects of HCCMF on tumors was evaluated against that of RCF. The results showed that the tumors treated by HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism behind tumor-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is caused through CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of various cancers.
The tumors in those in the group that were unaffected to RF EMF, and were fed a standard mouse diet. The tumors in HCCMF HCCMF group were infected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were removed in cases of excessive burden.
The tumors of the three groups also showed different growth curves. The HCCMF-treated tumors saw a significant decrease in tumour size after eight weeks. However, the tumours treated with RCF didn't show reduction in size. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is common when tumors are exposed to RCF. It is possible that the necrosis was due to a lack of oxygen in the larger tumours.
In conclusion, the findings indicate an AM-RF EMF exhibits anticancer activity in vitro and in the vivo. A number of studies have proven that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is emf blocker that AM RF EMF produces these effects due to CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained effect on the development of HCC tumours in living tissue.