Whether you live in an apartment or house, or you just need to ensure that your house is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the easiest is to reduce the use of electronic devices. emf blocker is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another method to protect your home against EMF radiations is to install an RF shielding canopy. This is a cloth of net that has EMF shielding and is used to stop EMFs from entering rooms. Another option is to have your home fitted with an electrical enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to involve down-regulation of cancer stem cells. This could be the reason for the long-term responses seen in patients with advanced HCC. However, the reason behind AM RF EMF's effect in cancer patients isn't yet clear.
block emf of AM RF EMF on HCC tumor growth in vivo was studied in mice. The tumors were separated in three different groups. The first group was not exposed RF EMF. The second group was exposed to RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
emf blocker for tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated by CACNA1H which is a protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have more broader implications for diagnosis and treatment of a variety of cancers.
The tumors in the control group were not exposed EMF from radiofrequency, and fed a normal mouse diet. The tumours in the HCCMF group were infected with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumours that were treated using RCF showed no shrinkage. The difference was significant. The tumors treated by RCF had necrosis that is common when tumors are exposed to RCF. The possibility is that this necrosis was caused by an absence of oxygen in the more invasive tumours.
In conclusion, the findings indicate that AM RF EMF has anticancer activity in vitro and in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF causes these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting effect on the development of HCC tumors in the vivo.